Not the patient is suffering from Nelarabine Arranon heart failure. However, magnetic resonance imaging suggested a high Pr Prevalence of subclinical brain infarction in patients with heart failure, but anything similar rates k Nnte in patients with vascular disease, But at the same age without heart failure are present. The origin of Schlaganf Cases of heart failure is likely to be diversified. Some will and some will be due to cardioembolic zerebrovaskul Re disease, but atherosclerosis in the aortic arch may also be an important source of embolism. Age, AF, diabetes and atherosclerosis are h INDICATIVE risk factors for stroke and heart failure. Well-treated patients with heart failure are now unlikely that high blood pressure, and these, as well as improved management of AF have k nnte Explained Ren, why the rate of Schlaganf do Cases in patients with heart failure have decreased over the past 30 years. Patients with severe heart failure or left ventricular Rer systolic dysfunction may be at even gr Be eren risk for stroke. Most patients with heart failure, a form of thrombosis prophylaxis received, although, as tr Gt is to lower rates of stroke, uncertain. The risk of h Hemorrhagic stroke is increased by an antithrombotic agent ht. Deep vein thrombosis and pulmonary embolism are rare diagnoses in ambulatory patients with heart failure and replace the modest effects in bettl Gerige patients due to worsening heart failure or concomitant diseases. The proportion of pl relooking Todesf ll By pulmonary embolism is difficult to quantify, but is generally regarded as unusual. Ultimately k can Two hours Ufigsten manifestations of vascular Ren events in heart failure in a deterioration of cardiac function and pl relooking to be death. Clinical guidelines for antithrombotic therapy in heart failure clinical guidelines in North America and Europe is an advocate of anticoagulation in patients with atrial fibrillation, but the lawyer not to use aspirin for CAD in patients with heart failure. Europ Ical guidelines explicitly define, there is no evidence that antiplatelet agents reduce the risk of atherosclerosis in patients with heart failure and r not The hardness Ratings For anticoagulation in patients other heart failure, au It in those with a prosthetic valve or AF. U.S. guidelines specifically in patients who have heart attacks and angina pectoris, the RF have suffered, but not the use of ACE inhibitors and beta blockers k Can also reduce the risk of heart attack and re-death, but it is less clear whether these patients by the use of aspirin or revascularization benefit. Other antiplatelet agents may not interact negatively with ACE inhibitors and k Can better effects in Pr Prevention of clinical events have, but their R Ability, positive impact on the outcome of HF has not been established. Observational studies and mechanistic antiplatelet agents aspirin in heart failure is the agent on the hour Ufigsten used antithrombotic agent in patients with heart failure in sinus rhythm. Although aspirin is platelet activation reduce heart failure, it may also reduce vascular Ren prostaglandin production leads to an increase in vascular Ren resistance, sodium retention, worsening renal function and reduced defense against vascular Re Pl Ttchenadh recession . In addition, k Can inhibitors of angiotensin-converting-enzyme part of their profits due to the increased Hten synthesis of prostaglandins and aspirin can practice with this st Ren. Even small doses of the form.